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Adenosine‐Sensitive Atrial Reentrant Tachycardia Originating from the Atrioventricular Nodal Transitional Area

Identifieur interne : 002059 ( Main/Exploration ); précédent : 002058; suivant : 002060

Adenosine‐Sensitive Atrial Reentrant Tachycardia Originating from the Atrioventricular Nodal Transitional Area

Auteurs : Yoshito Iesaka ; Atsushi Takahashi ; Masahiko Goya ; Yohkoh Soejima ; Yoshihiro Okamoto ; Hideomi Fujiwara ; Kazutaka Aonuma [Japon] ; Akihtko Nogami [Japon] ; Michiaki Hiroe [Japon] ; Fumiaki Marumo [Japon] ; Masayasu Hiraoka [Japon]

Source :

RBID : ISTEX:A13298A9AF7CE25CE1EF1419318EFA0FA490FC62

English descriptors

Abstract

Adenosine‐Sensitive AT from AVN Area. Introduction: Atrial tachycardia shows wide variations in its electrophysiologic properties and sites of origin. We report an atrial tachycardia with ECG manifestations and electrophysiologic characteristics similar to an atypical form of AV nodal reentrant tachycardia (AVNRT). Methods and Results: This supraventricular tachycardia was observed in 11 patients. It was initiated by atrial extrastimulation with an inverse relationship between the coupling interval of an extrastimulus and the postextrastimulus interval. Its induction was not related to a jump in the AH interval, and its perpetuation was independent of conduction block in the AV node. Ventricular pacing during tachycardia demonstrated AV dissociation without affecting the atrial cycle length. A very small dose of adenosine triphosphate (mean 3.9 ± 1.2 mg) could terminate the tachycardia. The earliest atrial activation during tachycardia was recorded at the low anteroseptal right atrium with a different intra‐atrial activation sequence from that recorded during ventricular pacing, where the tachycardia was successfully ablated in 9 of 10 attempted patients. Bidirectional AV nodal conduction remained unatttched after successful ablation. Conclusion: There may he an entity of adenosine‐sensitive atrial tachycardia probably due to focal reentry within the AV node or its transitional tissues without involvement of the AV nodal pathways. This tachycardia can he ablated without disturbing AV nodal conduction from the right atrial septum.

Url:
DOI: 10.1111/j.1540-8167.1997.tb00846.x


Affiliations:


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Le document en format XML

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